SBA is able to disrupt small intestinal metabolism and damage small intestinal villi via the ability of lectins to bind with brush border surfaces in the distal part of small intestine.
2.
Added difficulties for diagnosis are the fact that serological markers ( anti-tissue transglutaminase [ TG2 ] ) are not always present and many patients may have minor mucosal lesions, without atrophy of the intestinal villi.
3.
In untreated coeliac disease, villous atrophy is more common in children younger than three years, but in older children and adults, it is common to find minor intestinal lesions ( duodenal lymphocytosis ) with normal intestinal villi.
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